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Gum disease

Encyclopedia of Alternative Medicine by Patience Paradox

Definition

Gum disease is also called periodontal disease. It is defined as the inflammation of the structures that surround and support the teeth. If left untreated, gum disease may progress to the point where there is destruction of the jawbone. It is one of the most common causes of tooth loss. Periodontal disease is also a risk factor for coronary heart disease and preterm low birth weight.

Description

Gingivitis is the earliest stage of a gum infection. It may recur or even become chronic. If gingivitis is not treated properly, it may progress to periodontitis, an inflammation of the periodontal ligament that helps hold the teeth in the bone. Periodontitis is sometimes called pyorrhea, which means a pus discharge. Severe cases of periodontitis may affect the jawbone. A severe case of gum disease that comes on suddenly is a disease known as trench mouth. Trench mouth, or necrotizing ulcerative gingivitis, is also known as Vincent's infection. It is caused by an infection of both spirochetes and fusiform bacilli. It was once a major problem for soldiers during World War I. Currently, trench mouth is particularly common among teenagers and young adults under stress, often at examination time. Inflammation from trench mouth can also spread to nearby tissues of the face and neck.

Causes & symptoms

By far, the most common cause of gum disease is poor dental hygiene. Regular daily brushing and flossing of the teeth generally clears away food and bacteria buildup in the mouth. If the teeth are neglected, bacteria collect, and plaque forms on the teeth and gums. If the plaque is not removed, it mixes with saliva and hardens into tartar. Tartar irritates the gums and causes them to shrink away from the teeth, opening up spaces where more bacteria and plaque can collect. This cycle encourages increasingly severe inflammation and infection.

The mechanisms by which bacteria cause tissue destruction in advanced gum disease are not fully understood. Several bacterial products that diffuse through tissue are thought to play a role in gum disease. Toxins produced by some bacteria can kill cells. Studies show that the amount of endotoxin present correlates with the severity of periodontal disease. Other bacterial products include proteolytic enzymes, which are molecules that digest protein found in cells, thereby causing cell destruction. The human immune response has also been implicated in tissue destruction. As part of a normal immune response, white blood cells enter regions of inflammation to destroy bacteria. In the process of destroying bacteria, periodontal tissue is also destroyed.

Other factors can contribute to the development of gum disease. Smokers are more than two times as likely as nonsmokers to develop gum disease. Hormone levels contribute to the development of bacteria in the mouth. Thus pregnancy, puberty, menopause, and the use of oral or injectable contraceptives may create a climate that favors the development of gum disease. Additional factors include diabetes mellitus , scurvy, pellagra, allergies, leukemia, Crohn's disease , AIDS, chemotherapy, nutritional deficiencies, hydrochloric acid deficiency, poorly fitted fillings, radiation treatments, and exposure to heavy metals (mercury, lead, arsenic, and nickel). Medications that may contribute to the development of gum disease include phenytoin for controlling seizures; cyclosporine, which is taken by people who have had organ transplants; and the calcium channel blockers used to control blood pressure and heartbeat.

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The main symptoms of gingivitis are dark red swollen gums that are mushy and bleed easily. Pain is usually minimal. People with periodontitis have the same symptoms. In addition they may also have pain, loose teeth, and bad breath . Abscesses and pus may develop. The symptoms of trench mouth include sudden onset of illness accompanied by pain, bleeding gums, bad breath, and a grayish mucus that covers the gums.

Diagnosis

A dental examination and history will be taken. As the disease progresses, a dentist will be able to find hollowed pockets near the gums. X rays may be used to reveal the breakdown of bone. A smear of the gum area may be taken to determine the existence of any bacterial infections. The visualization of spirochetes can be used to confirm the diagnosis of trench mouth.

Treatment

Naturopathic treatment

Zinc, copper, folic acid , vitamin E , selenium, and vitamin A or beta-carotene are very helpful in slowing the progression of gum disease, especially if the patient has dietary deficiencies. Daily CoQ10 supplementation is also recommended. Mouthwashes that contain either a 0.1% folate solution or a 5% solution of zinc, or both, swished in the mouth and held there for at least a minute, can be taken twice daily.

Flavonoids help reduce inflammation and strengthen the gum tissue. A daily menu that includes foods rich in flavonoids is recommended. These foods include blueberries, hawthorn berries, onions, and grapes. Extracts of these foods may be used as well.

Homeopathy

The main homeopathic remedy for mild gum disease is Mercurius solubilis hahnemanni in a 6c potency. Natrum muriaticum, also in a 6c potency, is for more severe disease, especially if there is formation of pus. Homeopathic remedies can be taken four times daily for up to three days.

Ayurvedic medicine

Ayurvedic practitioners recommend a daily cup of water with the juice of a fresh lemon squeezed into it for bleeding gums. Five grams of amla powder in a cup of water daily is also recommended. The teeth can be brushed with catechu or neem powder or both.

Traditional Chinese medicine

According to traditional Chinese medicine , the gums are nourished by the liver's function. If the level of toxins in the body exceeds the liver's blood-cleansing limits, eventually the gums (and other parts of the body) become a breeding ground for disease. The Chinese also consider the liver as a reservoir of blood; it ensures that adequate blood and Qi (vital energy) are delivered to the muscles, gums, and joints.