Blood lead levels and sexual maturation in U.S. girls: the Third National Health and Nutrition Examination Survey, 1988-1994 - Children's Health

Environmental Health Perspectives, May, 2003 by Tiejian Wu, Germaine M. Buck, Pauline Mendola

Miller DT, Paschal DC, Gunter EW, Stoud PE, D'Angelo J, 1987, Determination of blood lead with electrothermal atomic absorption using a L'vov platform and matrix modifier. Analyst 112:1701.

National Center for Health Statistics. 1994. Plan and operation of the Third National Health and Nutrition Examination Survey, 1988-1994. Vital Health Stat 1(32).

--. 1998. NHANES III Household Youth Data File Documentation. National Health and Nutrition Examination Survey III, 1988-1994. (CD-ROM Series 11), Hyattsville, MD:Centers for Disease Control and Prevention.

National Research Council, 2000, Scientific Frontiers in Developmental Toxicology and Risk Assessment. Washington, DC:National Academy Press.

Nriagu JO. 1983. Saturine gout among Roman aristocrats: did lead poisoning contribute to the fall of the Empire? N Engl J Med 308:660-663.

O'Flaherty EJ, 1995. Physiologically based models for bone-seeking elements. V. Lead absorption and disposition in childhood. Toxicol Appl Pharmacol 131:297-308.

Pirkle JL, Brodie DJ, Gunter EW, et al, 1994. The decline in blood lead levels in the United States: the National Health and Nutrition Examination Surveys (NHANES). JAMA 272(4):284-291,

Rodamilans M, Osaba MJM, To-Figueras J, Fillat FR, Marques JM, Perez P, Corbella J. 1988. Lead toxicity on endocrine testicular function in ah occupational exposed population, Hum Toxicol 7:125-128.

Ronis MJJ, Badger TM, Shema SJ, Roberson PK. 1998a. Endocrine mechanisms underlying the growth effects of developmental lead exposure in the rat. J Toxicol Environ Health 54:101-120.

Ronis MJJ, Badger TM, Shema SJ, Roberson PK, Sjaikh F. 1998b. Effects on pubertal growth in rats exposed to lead perinatally or continuously through development. J Toxicol Environ Health 53:327-341.

Ronis MJJ, Gandy J, Badger T. 1998c. Endocrine mechanisms underlying reproductive toxicity in the developing rat chronically exposed to dietary lead. J Toxicol Environ Health 54:77-99.

SAS. 1992. SAS/STAT User's Guide. Version 6, 4th ed. Cary, NC:SAS Institute, Inc.

Six KM, Goyer RA. 1972. The influence of iron deficiency on tissue content and toxicity of ingested lead in the rat. J Lab Clin Med 79:128-136.

Sokol RZ, Wang S, Wan YY, Stanczyk FZ, Gentzscheir E, Chapin REI. 2002. Long-term, low-dose lead exposure alters the gonadotropin-releasing hormone system in the male rat. Environ Health Perspect 110:871-874.

SUDAAN. 1995, User's Manual. Release 6.40. Chapel Hill, NC:Research Triangle Institute.

Tanner JM, 1986. Normal growth and techniques of growth assessment. Clin Endocrinol Metab 15:411-451.

Treloar SA, Martin NG. 1990. Age at menarche as a fitness trait: nonadditive genetic variance detected in a large twin sample, Am J Hum Genet 47:137-148.

U.S. EPA. 1994. Guidance Manual for the Integrated Exposure Uptake Model for Lead in Children. National Technical Information Service. PB 93-963510. Washington, DC:U.S. Environmental Protection Agency.

Vivoli G, Fantuzzi G, Bergomi M, Tonelli E, Gatto MR, Zanetti F, et al. 1993, Relationship between low lead exposure and somatic growth in adolescents. J Expo Anal Environ Epidemiol 3(suppl 3):201-209.


 

BNET TalkbackShare your ideas and expertise on this topic

Please add your comment:

  1. You are currently: a Guest |
  2.  

Basic HTML tags that work in comments are: bold (<b></b>), italic (<i></i>), underline (<u></u>), and hyperlink (<a href></a)

advertisement
Click Here
advertisement
  • Click Here
  • Click Here
  • Click Here
advertisement

Content provided in partnership with Thompson Gale